Friday, November 29, 2019

The favorite drink of Inspector Maigret in Georges Simenon s âàðèàíò â detective novels âàðèà free essay sample

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Food is one of the human organic structures sources of energy ( # 1074 ; # 1072 ; # 1088 ; # 1080 ; # 1072 ; # 1085 ; # 1090 ; # 1074 ; ) . 2. The favorite drink of Inspector MaigretIn Georges Simenon s( # 1074 ; # 1072 ; # 1088 ; # 1080 ; # 1072 ; # 1085 ; # 1090 ; # 1074 ; ) detective novels ( # 1074 ; # 1072 ; # 1088 ; # 1080 ; # 1072 ; # 1085 ; # 1090 ; # 1073 ; ) is apple brandy Calvados . 3. They introduced the new devices ( # 1074 ; # 1072 ; # 1088 ; # 1080 ; # 1072 ; # 1085 ; # 1090 ; # 1072 ; ) into the procedure of production instantly. 4. Plant converts ( # 1074 ; # 1072 ; # 1088 ; # 1080 ; # 1072 ; # 1085 ; # 1090 ; # 1072 ; ) solar energy to nutrient by photosynthesis. # 1054 ; # 1090 ; # 1074 ; # 1077 ; # 1090 ;: 1. # 1055 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1077 ; # 1086 ; # 1076 ; # 1080 ; # 1085 ; # 1080 ; # 1079 ; # 1080 ; # 1089 ; # 1090 ; # 1086 ; # 1095 ; # 1085 ; # 1080 ; # 1082 ; # 1086 ; # 1074 ; # 1101 ; # 1085 ; # 1077 ; # 1088 ; # 1075 ; # 1080 ; # 1080 ; # 1095 ; # 1077 ; # 1083 ; # 1086 ; # 1074 ; # 1077 ; # 1082 ; # 1072 ; . 2. # 1051 ; # 1102 ; # 1073 ; # 1080 ; # 1084 ; # 1099 ; # 1081 ; # 1085 ; # 1072 ; # 1087 ; # 1080 ; # 1090 ; # 1086 ; # 1082 ; # 1048 ; # 1085 ; # 1089 ; # 1087 ; # 1077 ; # 1082 ; # 1090 ; # 1086 ; # 1088 ; # 1072 ; # 1052 ; # 1101 ; # 1081 ; # 1075 ; # 1088 ; # 1077 ; # 1090 ; # 1072 ; # 1074 ; # 1076 ; # 1077 ; # 1090 ; # 1077 ; # 1082 ; # 1090 ; # 1080 ; # 1074 ; # 1085 ; # 1099 ; # 1093 ; # 1088 ; # 1086 ; # 1084 ; # 1072 ; # 1085 ; # 1072 ; # 1093 ; # 1044 ; # 1078 ; # 1086 ; # 1088 ; # 1076 ; # 1078 ; # 1072 ; # 1057 ; # 1080 ; # 1084 ; # 1077 ; # 1085 ; # 1086 ; # 1085 ; # 1072 ; # 1103 ; # 1073 ; # 1083 ; # 1086 ; # 1095 ; # 1085 ; # 1099 ; # 1081 ; # 1073 ; # 1088 ; # 1077 ; # 1085 ; # 1076 ; # 1080 ; # 1050 ; # 1072 ; # 1083 ; # 1100 ; # 1074 ; # 1072 ; # 1076 ; # 1086 ; # 1089 ; . 3. # 1054 ; # 1085 ; # 1080 ; # 1085 ; # 1077 ; # 1079 ; # 1072 ; # 1084 ; # 1077 ; # 1076 ; # 1083 ; # 1080 ; # 1090 ; # 1077 ; # 1083 ; # 1100 ; # 1085 ; # 1086 ; # 1074 ; # 1074 ; # 1086 ; # 1076 ; # 1080 ; # 1083 ; # 1080 ; # 1085 ; # 1086 ; # 1074 ; # 1099 ; # 1077 ; # 1091 ; # 1089 ; # 1090 ; # 1088 ; # 1086 ; # 1081 ; # 1089 ; # 1090 ; # 1074 ; # 1072 ; # 1074 ; # 1087 ; # 1088 ; # 1086 ; # 1094 ; # 1077 ; # 1089 ; # 1089 ; # 1087 ; # 1088 ; # 1086 ; # 1080 ; # 1079 ; # 1074 ; # 1086 ; # 1076 ; # 1089 ; # 1090 ; # 1074 ; # 1072 ; . 4. # 1056 ; # 1072 ; # 1089 ; # 1090 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; # 1082 ; # 1086 ; # 1085 ; # 1074 ; # 1077 ; # 1088 ; # 1090 ; # 1080 ; # 1088 ; # 1091 ; # 1077 ; # 1090 ; # 1089 ; # 1086 ; # 1083 ; # 1085 ; # 1077 ; # 1095 ; # 1085 ; # 1091 ; # 1102 ; # 1101 ; # 1085 ; # 1077 ; # 1088 ; # 1075 ; # 1080 ; # 1102 ; # 1082 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1102 ; # 1092 ; # 1086 ; # 1090 ; # 1086 ; # 1089 ; # 1080 ; # 1085 ; # 1090 ; # 1077 ; # 1079 ; # 1086 ; # 1084 ; . 2. # 1055 ; # 1077 ; # 1088 ; # 1077 ; # 1087 ; # 1080 ; # 1096 ; # 1080 ; # 1090 ; # 1077 ; # 1089 ; # 1083 ; # 1077 ; # 1076 ; # 1091 ; # 1102 ; # 1097 ; # 1080 ; # 1077 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1083 ; # 1086 ; # 1078 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1080 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1077 ; # 1076 ; # 1080 ; # 1090 ; # 1077 ; # 1080 ; # 1093 ; , # 1091 ; # 1095 ; # 1080 ; # 1090 ; # 1099 ; # 1074 ; # 1072 ; # 1103 ; # 1086 ; # 1089 ; # 1086 ; # 1073 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1089 ; # 1090 ; # 1080 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1086 ; # 1076 ; # 1072 ; # 1085 ; # 1072 ; # 1088 ; # 1091 ; # 1089 ; # 1089 ; # 1082 ; # 1080 ; # 1081 ; # 1103 ; # 1079 ; # 1099 ; # 1082 ; # 1086 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1077 ; # 1083 ; # 1077 ; # 1085 ; # 1080 ; # 1081 ; , # 1074 ; # 1099 ; # 1088 ; # 1072 ; # 1078 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1093 ; # 1080 ; # 1084 ; # 1077 ; # 1085 ; # 1077 ; # 1084 ; # 1089 ; # 1091 ; # 1097 ; # 1077 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1090 ; # 1077 ; # 1083 ; # 1100 ; # 1085 ; # 1099 ; # 1084 ; . 1. Protein combinations such as oats and nuts in mueslisupply really high quality protein. 2. Industrial Revolution enforced a extremist alteration in the nutrient wonts. 3. One nutrient supplies both energy and edifice stuff, another regulates organic structure procedures and gives energy. # 1054 ; # 1090 ; # 1074 ; # 1077 ; # 1090 ;: 1. # 1050 ; # 1086 ; # 1084 ; # 1073 ; # 1080 ; # 1085 ; # 1072 ; # 1094 ; # 1080 ; # 1080 ; # 1073 ; # 1077 ; # 1083 ; # 1082 ; # 1072 ; # 1090 ; # 1080 ; # 1087 ; # 1072 ; # 1086 ; # 1074 ; # 1089 ; # 1072 ; # 1080 ; # 1086 ; # 1088 ; # 1077 ; # 1093 ; # 1086 ; # 1074 ; # 1074 ; # 1084 ; # 1102 ; # 1089 ; # 1083 ; # 1080 ; # 1086 ; # 1073 ; # 1077 ; # 1089 ; # 1087 ; # 1077 ; # 1095 ; # 1080 ; # 1074 ; # 1072 ; # 1102 ; # 1090 ; # 1086 ; # 1095 ; # 1077 ; # 1085 ; # 1100 ; # 1074 ; # 1099 ; # 1089 ; # 1086 ; # 1082 ; # 1086 ; # 1082 ; # 1072 ; # 1095 ; # 1077 ; # 1089 ; # 1090 ; # 1074 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1081 ; # 1073 ; # 1077 ; # 1083 ; # 1086 ; # 1082 ; . 2. # 1055 ; # 1088 ; # 1086 ; # 1084 ; # 1099 ; # 1096 ; # 1083 ; # 1077 ; # 1085 ; # 1085 ; # 1072 ; # 1103 ; # 1056 ; # 1077 ; # 1074 ; # 1086 ; # 1083 ; # 1102 ; # 1094 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1087 ; # 1080 ; # 1089 ; # 1072 ; # 1083 ; # 1072 ; # 1088 ; # 1072 ; # 1076 ; # 1080 ; # 1082 ; # 1072 ; # 1083 ; # 1100 ; # 1085 ; # 1091 ; # 1102 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1084 ; # 1077 ; # 1085 ; # 1091 ; # 1074 ; # 1087 ; # 1088 ; # 1080 ; # 1074 ; # 1099 ; # 1095 ; # 1082 ; # 1072 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; . 3. # 1054 ; # 1076 ; # 1080 ; # 1085 ; # 1074 ; # 1080 ; # 1076 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; # 1089 ; # 1085 ; # 1072 ; # 1073 ; # 1078 ; # 1072 ; # 1077 ; # 1090 ; # 1080 ; # 1101 ; # 1085 ; # 1077 ; # 1088 ; # 1075 ; # 1080 ; # 1077 ; # 1081 ; # 1080 ; # 1089 ; # 1090 ; # 1088 ; # 1086 ; # 1080 ; # 1090 ; # 1077 ; # 1083 ; # 1100 ; # 1085 ; # 1099 ; # 1084 ; # 1084 ; # 1072 ; # 1090 ; # 1077 ; # 1088 ; # 1080 ; # 1072 ; # 1083 ; # 1086 ; # 1084 ; , # 1076 ; # 1088 ; # 1091 ; # 1075 ; # 1086 ; # 1081 ; # 1088 ; # 1077 ; # 1075 ; # 1091 ; # 1083 ; # 1080 ; # 1088 ; # 1091 ; # 1077 ; # 1090 ; # 1086 ; # 1073 ; # 1084 ; # 1077 ; # 1085 ; , # 1086 ; # 1073 ; # 1088 ; # 1072 ; # 1073 ; # 1072 ; # 1090 ; # 1099 ; # 1074 ; # 1072 ; # 1077 ; # 1090 ; # 1080 ; # 1076 ; # 1072 ; # 1077 ; # 1090 ; # 1101 ; # 1085 ; # 1077 ; # 1088 ; # 1075 ; # 1080 ; # 1102 ; . 3. # 1055 ; # 1077 ; # 1088 ; # 1077 ; # 1087 ; # 1080 ; # 1096 ; # 1080 ; # 1090 ; # 1077 ; # 1089 ; # 1083 ; # 1077 ; # 1076 ; # 1091 ; # 1102 ; # 1097 ; # 1080 ; # 1077 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1083 ; # 1086 ; # 1078 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; , # 1089 ; # 1086 ; # 1076 ; # 1077 ; # 1088 ; # 1078 ; # 1072 ; # 1097 ; # 1080 ; # 1077 ; # 1088 ; # 1072 ; # 1079 ; # 1085 ; # 1099 ; # 1077 ; # 1092 ; # 1086 ; # 1088 ; # 1084 ; # 1099 ; # 1089 ; # 1088 ; # 1072 ; # 1074 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; , # 1080 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1077 ; # 1076 ; # 1080 ; # 1090 ; # 1077 ; # 1080 ; # 1093 ; # 1085 ; # 1072 ; # 1088 ; # 1091 ; # 1089 ; # 1089 ; # 1082 ; # 1080 ; # 1081 ; # 1103 ; # 1079 ; # 1099 ; # 1082 ; . 1. This job is much more serious than that one. 2. The natural toxicants are as harmful for wellness as the chemical 1s. 3. The more you read about nutrient, the better you know about its belongingss. 4. The myocardial infarctioncrobiologist s minuteT of import part to the nutrient Indusattempt isthe production of safe merchandises. # 1054 ; # 1090 ; # 1074 ; # 1077 ; # 1090 ;: 1. # 1069 ; # 1090 ; # 1072 ; # 1087 ; # 1088 ; # 1086 ; # 1073 ; # 1083 ; # 1077 ; # 1084 ; # 1072 ; # 1085 ; # 1072 ; # 1084 ; # 1085 ; # 1086 ; # 1075 ; # 1086 ; # 1089 ; # 1077 ; # 1088 ; # 1100 ; # 1077 ; # 1079 ; # 1085 ; # 1077 ; # 1081 ; , # 1095 ; # 1077 ; # 1084 ; # 1082 ; # 1072 ; # 1082 ; # 1072 ; # 1103 ; # 1073 ; # 1099 ; # 1090 ; # 1086 ; # 1085 ; # 1080 ; # 1073 ; # 1099 ; # 1083 ; # 1072 ; . 2. # 1045 ; # 1089 ; # 1090 ; # 1077 ; # 1089 ; # 1090 ; # 1074 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1077 ; # 1103 ; # 1076 ; # 1099 ; # 1089 ; # 1090 ; # 1086 ; # 1083 ; # 1100 ; # 1078 ; # 1077 ; # 1074 ; # 1088 ; # 1077 ; # 1076 ; # 1085 ; # 1099 ; # 1076 ; # 1083 ; # 1103 ; # 1079 ; # 1076 ; # 1086 ; # 1088 ; # 1086 ; # 1074 ; # 1100 ; # 1103 ; # 1082 ; # 1072 ; # 1082 ; # 1080 ; # 1093 ; # 1080 ; # 1084 ; # 1080 ; # 1095 ; # 1077 ; # 1089 ; # 1082 ; # 1080 ; # 1077 ; . 3. # 1063 ; # 1077 ; # 1084 ; # 1073 ; # 1086 ; # 1083 ; # 1100 ; # 1096 ; # 1077 ; # 1042 ; # 1099 ; # 1095 ; # 1080 ; # 1090 ; # 1072 ; # 1077 ; # 1090 ; # 1077 ; # 1086 ; # 1077 ; # 1076 ; # 1077 ; , # 1090 ; # 1077 ; # 1084 ; # 1083 ; # 1091 ; # 1095 ; # 1096 ; # 1077 ; # 1042 ; # 1099 ; # 1079 ; # 1085 ; # 1072 ; # 1077 ; # 1090 ; # 1077 ; # 1086 ; # 1077 ; # 1075 ; # 1086 ; # 1089 ; # 1074 ; # 1086 ; # 1081 ; # 1089 ; # 1090 ; # 1074 ; # 1072 ; # 1093 ; . 4. # 1057 ; # 1072 ; # 1084 ; # 1099 ; # 1081 ; # 1074 ; # 1072 ; # 1078 ; # 1085 ; # 1099 ; # 1081 ; # 1074 ; # 1082 ; # 1083 ; # 1072 ; # 1076 ; # 1084 ; # 1080 ; # 1082 ; # 1088 ; # 1086 ; # 1073 ; # 1080 ; # 1086 ; # 1083 ; # 1086 ; # 1075 ; # 1072 ; # 1074 ; # 1087 ; # 1080 ; # 1097 ; # 1077 ; # 1074 ; # 1091 ; # 1102 ; # 1087 ; # 1088 ; # 1086 ; # 1084 ; # 1099 ; # 1096 ; # 1083 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1089 ; # 1090 ; # 1100 ; # 1087 ; # 1088 ; # 1086 ; # 1080 ; # 1079 ; # 1074 ; # 1086 ; # 1076 ; # 1089 ; # 1090 ; # 1074 ; # 1086 ; # 1073 ; # 1077 ; # 1079 ; # 1086 ; # 1087 ; # 1072 ; # 1089 ; # 1085 ; # 1099 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1091 ; # 1082 ; # 1090 ; # 1086 ; # 1074 ; . 4. # 1055 ; # 1077 ; # 1088 ; # 1077 ; # 1087 ; # 1080 ; # 1096 ; # 1080 ; # 1090 ; # 1077 ; # 1080 ; # 1087 ; # 1080 ; # 1089 ; # 1100 ; # 1084 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1077 ; # 1076 ; # 1080 ; # 1090 ; # 1077 ; # 1085 ; # 1072 ; # 1088 ; # 1091 ; # 1089 ; # 1089 ; # 1082 ; # 1080 ; # 1081 ; # 1103 ; # 1079 ; # 1099 ; # 1082 ; # 1089 ; # 1083 ; # 1077 ; # 1076 ; # 1091 ; # 1102 ; # 1097 ; # 1080 ; # 1077 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1083 ; # 1086 ; # 1078 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; , # 1086 ; # 1073 ; # 1088 ; # 1072 ; # 1097 ; # 1072 ; # 1103 ; # 1086 ; # 1089 ; # 1086 ; # 1073 ; # 1086 ; # 1077 ; # 1074 ; # 1085 ; # 1080 ; # 1084 ; # 1072 ; # 1085 ; # 1080 ; # 1077 ; # 1085 ; # 1072 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1086 ; # 1076 ; # 1085 ; # 1077 ; # 1086 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1077 ; # 1083 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1093 ; # 1084 ; # 1077 ; # 1089 ; # 1090 ; # 1086 ; # 1080 ; # 1084 ; # 1077 ; # 1085 ; # 1080 ; # 1081 ; . 1. There are some major procedures for nutrient saving. 2. Nitrogeno one wanted to eat nutrient WisconsinThursday chemical preservatidegree Fahrenheits and addiTidegree Fahrenheits. 3. Are at that place any colouring agents in cosmetics, drugs? 4. Bites are popular. Peoples eat them anyplace at any Time. # 1054 ; # 1090 ; # 1074 ; # 1077 ; # 1090 ;: 1. # 1045 ; # 1089 ; # 1090 ; # 1100 ; # 1085 ; # 1077 ; # 1082 ; # 1086 ; # 1090 ; # 1086 ; # 1088 ; # 1099 ; # 1077 ; # 1075 ; # 1083 ; # 1072 ; # 1074 ; # 1085 ; # 1099 ; # 1077 ; # 1087 ; # 1088 ; # 1086 ; # 1094 ; # 1077 ; # 1089 ; # 1089 ; # 1099 ; # 1076 ; # 1083 ; # 1103 ; # 1089 ; # 1086 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; . 2. # 1053 ; # 1080 ; # 1082 ; # 1090 ; # 1086 ; # 1085 ; # 1077 ; # 1093 ; # 1086 ; # 1090 ; # 1077 ; # 1083 ; # 1073 ; # 1099 ; # 1077 ; # 1089 ; # 1090 ; # 1100 ; # 1077 ; # 1076 ; # 1091 ; # 1089 ; # 1093 ; # 1080 ; # 1084 ; # 1080 ; # 1095 ; # 1077 ; # 1089 ; # 1082 ; # 1080 ; # 1084 ; # 1080 ; # 1082 ; # 1086 ; # 1085 ; # 1089 ; # 1077 ; # 1088 ; # 1074 ; # 1072 ; # 1085 ; # 1090 ; # 1072 ; # 1084 ; # 1080 ; # 1080 ; # 1076 ; # 1086 ; # 1073 ; # 1072 ; # 1074 ; # 1082 ; # 1072 ; # 1084 ; # 1080 ; . 3. # 1045 ; # 1089 ; # 1090 ; # 1100 ; # 1083 ; # 1080 ; # 1093 ; # 1080 ; # 1084 ; # 1080 ; # 1095 ; # 1077 ; # 1089 ; # 1082 ; # 1080 ; # 1077 ; # 1101 ; # 1083 ; # 1077 ; # 1084 ; # 1077 ; # 1085 ; # 1090 ; # 1099 ; # 1074 ; # 1082 ; # 1086 ; # 1089 ; # 1084 ; # 1077 ; # 1090 ; # 1080 ; # 1082 ; # 1077 ; , # 1083 ; # 1077 ; # 1082 ; # 1072 ; # 1088 ; # 1089 ; # 1090 ; # 1074 ; # 1072 ; # 1093 ; ? 4. # 1047 ; # 1072 ; # 1082 ; # 1091 ; # 1089 ; # 1082 ; # 1080 ; # 1087 ; # 1086 ; # 1087 ; # 1091 ; # 1083 ; # 1103 ; # 1088 ; # 1085 ; # 1099 ; . # 1051 ; # 1102 ; # 1076 ; # 1080 ; # 1077 ; # 1076 ; # 1103 ; # 1090 ; # 1080 ; # 1093 ; # 1087 ; # 1086 ; # 1074 ; # 1089 ; # 1102 ; # 1076 ; # 1091 ; # 1074 ; # 1083 ; # 1102 ; # 1073 ; # 1086 ; # 1077 ; # 1074 ; # 1088 ; # 1077 ; # 1084 ; # 1103 ; . 5. # 1055 ; # 1077 ; # 1088 ; # 1077 ; # 1087 ; # 1080 ; # 1096 ; # 1080 ; # 1090 ; # 1077 ; # 1089 ; # 1083 ; # 1077 ; # 1076 ; # 1091 ; # 1102 ; # 1097 ; # 1080 ; # 1077 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1083 ; # 1086 ; # 1078 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; , # 1086 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1077 ; # 1083 ; # 1080 ; # 1090 ; # 1077 ; # 1074 ; # 1085 ; # 1080 ; # 1093 ; # 1074 ; # 1080 ; # 1076 ; # 1086 ; # 1074 ; # 1088 ; # 1077 ; # 1084 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1077 ; # 1092 ; # 1086 ; # 1088 ; # 1084 ; # 1099 ; # 1075 ; # 1083 ; # 1072 ; # 1075 ; # 1086 ; # 1083 ; # 1086 ; # 1074 ; # 1080 ; # 1091 ; # 1082 ; # 1072 ; # 1078 ; # 1080 ; # 1090 ; # 1077 ; # 1080 ; # 1093 ; # 1080 ; # 1085 ; # 1092 ; # 1080 ; # 1085 ; # 1080 ; # 1090 ; # 1080 ; # 1074 ; ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1077 ; # 1076 ; # 1080 ; # 1090 ; # 1077 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1083 ; # 1086 ; # 1078 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1085 ; # 1072 ; # 1088 ; # 1091 ; # 1089 ; # 1089 ; # 1082 ; # 1080 ; # 1081 ; # 1103 ; # 1079 ; # 1099 ; # 1082 ; . 1.Modern methods demonstrated( Past Indefinite Active Voice ) the toxic effects of some coloring material ingredients. 2. You wbadly look into( Future Continuous Passive Voice ) the operation of generators, thrusts, electrical measurement instruments and the similar. 3. Carbohydrate is( Present Continuous Active Voice ) a signifier of sugar. 4. This alteration in works and carnal nutrients took( Past Indefinite Active Voice ) topographic point bit by bit over many centuries. # 1054 ; # 1090 ; # 1074 ; # 1077 ; # 1090 ;: 1. # 1057 ; # 1086 ; # 1074 ; # 1088 ; # 1077 ; # 1084 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1077 ; # 1084 ; # 1077 ; # 1090 ; # 1086 ; # 1076 ; # 1099 ; # 1076 ; # 1077 ; # 1084 ; # 1086 ; # 1085 ; # 1089 ; # 1090 ; # 1088 ; # 1080 ; # 1088 ; # 1086 ; # 1074 ; # 1072 ; # 1083 ; # 1080 ; # 1103 ; # 1076 ; # 1086 ; # 1074 ; # 1080 ; # 1090 ; # 1099 ; # 1077 ; # 1101 ; # 1092 ; # 1092 ; # 1077 ; # 1082 ; # 1090 ; # 1099 ; # 1085 ; # 1077 ; # 1082 ; # 1086 ; # 1090 ; # 1086 ; # 1088 ; # 1099 ; # 1093 ; # 1094 ; # 1074 ; # 1077 ; # 1090 ; # 1086 ; # 1074 ; # 1099 ; # 1093 ; # 1082 ; # 1086 ; # 1084 ; # 1087 ; # 1086 ; # 1085 ; # 1077 ; # 1085 ; # 1090 ; # 1086 ; # 1074 ; . 2. Âà » à ¬Ãƒ ®Ãƒ ¦Ãƒ ¥Ãƒ ²Ãƒ ¥ à ¯Ãƒ °Ãƒ ®Ãƒ ¢Ãƒ ¥Ãƒ °Ãƒ ¨Ãƒ ²Ãƒ ¼ à ¤Ãƒ ¥Ãƒ ©Ãƒ ±Ãƒ ²Ãƒ ¢Ãƒ ¨Ãƒ ¥ à £Ãƒ ¥Ãƒ ­Ãƒ ¥Ãƒ °Ãƒ  Ãƒ ²Ãƒ ®Ãƒ °Ãƒ ®Ãƒ ¢ , à ¤Ãƒ ¢Ãƒ ¨Ãƒ £Ãƒ  Ãƒ ²Ãƒ ¥Ãƒ «Ãƒ ¥Ãƒ © , à ½Ãƒ «Ãƒ ¥Ãƒ ªÃƒ ²Ãƒ °Ãƒ ¨Ãƒ ·Ãƒ ¥Ãƒ ±Ãƒ ªÃƒ ¨Ãƒ µ à ¨Ãƒ §Ãƒ ¬Ãƒ ¥Ãƒ °Ãƒ ¨Ãƒ ²Ãƒ ¥Ãƒ «Ãƒ ¼Ãƒ ­Ãƒ »Ãƒ µ à ¯Ãƒ °Ãƒ ¨Ãƒ ¡Ãƒ ®Ãƒ °Ãƒ ®Ãƒ ¢ à ¨ à ².à ¯ . 3. # 1059 ; # 1075 ; # 1083 ; # 1077 ; # 1074 ; # 1086 ; # 1076 ; # 1092 ; # 1086 ; # 1088 ; # 1084 ; # 1072 ; # 1089 ; # 1072 ; # 1093 ; # 1072 ; # 1088 ; # 1072 ; . 4. # 1069 ; # 1090 ; # 1072 ; # 1084 ; # 1086 ; # 1076 ; # 1080 ; # 1092 ; # 1080 ; # 1082 ; # 1072 ; # 1094 ; # 1080 ; # 1103 ; # 1088 ; # 1072 ; # 1089 ; # 1090 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1080 ; # 1087 ; # 1080 ; # 1097 ; # 1077 ; # 1074 ; # 1099 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1091 ; # 1082 ; # 1090 ; # 1072 ; # 1093 ; # 1078 ; # 1080 ; # 1074 ; # 1086 ; # 1090 ; # 1085 ; # 1086 ; # 1075 ; # 1086 ; # 1087 ; # 1086 ; # 1089 ; # 1090 ; # 1077 ; # 1087 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1086 ; # 1073 ; # 1088 ; # 1077 ; # 1083 ; # 1072 ; # 1089 ; # 1074 ; # 1086 ; # 1077 ; # 1084 ; # 1077 ; # 1089 ; # 1090 ; # 1086 ; # 1079 ; # 1072 ; # 1084 ; # 1085 ; # 1086 ; # 1075 ; # 1080 ; # 1077 ; # 1089 ; # 1090 ; # 1086 ; # 1083 ; # 1077 ; # 1090 ; # 1080 ; # 1103 ; . 6. # 1055 ; # 1088 ; # 1086 ; # 1095 ; # 1090 ; # 1080 ; # 1090 ; # 1077 ; # 1080 ; # 1087 ; # 1080 ; # 1089 ; # 1100 ; # 1084 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1087 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1077 ; # 1076 ; # 1080 ; # 1090 ; # 1077 ; # 1085 ; # 1072 ; # 1088 ; # 1091 ; # 1089 ; # 1089 ; # 1082 ; # 1080 ; # 1081 ; # 1103 ; # 1079 ; # 1099 ; # 1082 ; # 1090 ; # 1077 ; # 1082 ; # 1089 ; # 1090 ; . FOOD. 1. Food is indispensable to the nutrition of any organic structure or any signifier of life. Food normally consists of protein, saccharide, and fat. The organic structure uses them to prolong growing, fix, and critical procedures and to supply energy. 2. A figure of research works present a description of the procedures of soaking up and use of nutrient. We get an information on such methods as fixing natural nutrients for cookery, ingestion, or storage. From the articles we besides come to cognize about the art of selecting, preparing, and functioning all right nutrients or nutrients traditional to a part or population. So nutrient takes an of import topographic point in the circle of acquisition. 3. From the scientists point of position nutrient saving is the most serious job. Any of a figure of methods of saving supports nutrient from spoilage after crop or slaughter. 4. The pattern of continuing nutrient has a prehistoric beginning. The ancient people dried fruits and veggies, parched cereal grains, salted and dried fish and game. These antique methods developed really easy and were strictly empirical-fermentation, drying, smoke, and bring arounding with salt. 5. Then rapid progresss followed. Thus we get an information about bacterium, casts, or barms, micro-organisms. They are the major causes of nutrient spoilage. But there are about seven celebrated well-known methods in nutrient saving now, such as: chilling or freeze, desiccation, canning, smoke, salting, candying, and the add-on of chemical preservatives and inhibitors.Very frequently several rules combine. 6. The modern aim of nutrient saving includes concern for nutrient quality, for economic system, and, particularly, for convenience in add-on to the bar of spoilage. Color or visual aspect, spirit, texture or consistence, and alimentary value are the major quality factors. Naturally, quality factors depend on the degree of economic development. # 1055 ; # 1077 ; # 1088 ; # 1077 ; # 1074 ; # 1086 ; # 1076 ;: # 1045 ; # 1076 ; # 1072 ; . 1. # 1045 ; # 1076 ; # 1072 ; # 8211 ; # 1078 ; # 1080 ; # 1079 ; # 1085 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1085 ; # 1077 ; # 1086 ; # 1073 ; # 1093 ; # 1086 ; # 1076 ; # 1080 ; # 1084 ; # 1072 ; # 1076 ; # 1083 ; # 1103 ; # 1083 ; # 1102 ; # 1073 ; # 1086 ; # 1075 ; # 1086 ; # 1086 ; # 1088 ; # 1075 ; # 1072 ; # 1085 ; # 1080 ; # 1079 ; # 1084 ; # 1072 ; # 1080 ; # 1083 ; # 1080 ; # 1083 ; # 1102 ; # 1073 ; # 1086 ; # 1081 ; # 1092 ; # 1086 ; # 1088 ; # 1084 ; # 1099 ; # 1078 ; # 1080 ; # 1079 ; # 1085 ; # 1080 ; . # 1055 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1077 ; # 1086 ; # 1073 ; # 1099 ; # 1095 ; # 1085 ; # 1086 ; # 1089 ; # 1086 ; # 1089 ; # 1090 ; # 1086 ; # 1080 ; # 1090 ; # 1080 ; # 1079 ; # 1073 ; # 1077 ; # 1083 ; # 1082 ; # 1072 ; , # 1091 ; # 1075 ; # 1083 ; # 1077 ; # 1074 ; # 1086 ; # 1076 ; # 1072 ; , # 1080 ; # 1078 ; # 1080 ; # 1088 ; # 1072 ; . # 1054 ; # 1088 ; # 1075 ; # 1072 ; # 1085 ; # 1080 ; # 1079 ; # 1084 ; # 1080 ; # 1089 ; # 1087 ; # 1086 ; # 1083 ; # 1100 ; # 1079 ; # 1091 ; # 1077 ; # 1090 ; # 1080 ; # 1093 ; , # 1095 ; # 1090 ; # 1086 ; # 1073 ; # 1099 ; # 1089 ; # 1087 ; # 1086 ; # 1089 ; # 1086 ; # 1073 ; # 1089 ; # 1090 ; # 1074 ; # 1086 ; # 1074 ; # 1072 ; # 1090 ; # 1100 ; # 1088 ; # 1086 ; # 1089 ; # 1090 ; # 1091 ; , # 1074 ; # 1086 ; # 1089 ; # 1089 ; # 1090 ; # 1072 ; # 1085 ; # 1086 ; # 1074 ; # 1083 ; # 1077 ; # 1085 ; # 1080 ; # 1102 ; # 1078 ; # 1080 ; # 1079 ; # 1085 ; # 1077 ; # 1085 ; # 1085 ; # 1099 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1094 ; # 1077 ; # 1089 ; # 1089 ; # 1086 ; # 1074 ; # 1080 ; # 1087 ; # 1086 ; # 1089 ; # 1090 ; # 1072 ; # 1074 ; # 1083 ; # 1103 ; # 1090 ; # 1100 ; # 1101 ; # 1085 ; # 1077 ; # 1088 ; # 1075 ; # 1080 ; # 1102 ; . 2. # 1052 ; # 1085 ; # 1086 ; # 1078 ; # 1077 ; # 1089 ; # 1090 ; # 1074 ; # 1086 ; # 1088 ; # 1072 ; # 1073 ; # 1086 ; # 1090 ; # 1080 ; # 1089 ; # 1089 ; # 1083 ; # 1077 ; # 1076 ; # 1086 ; # 1074 ; # 1072 ; # 1085 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1089 ; # 1090 ; # 1072 ; # 1074 ; # 1083 ; # 1103 ; # 1102 ; # 1090 ; # 1086 ; # 1087 ; # 1080 ; # 1089 ; # 1072 ; # 1085 ; # 1080 ; # 1077 ; # 1087 ; # 1088 ; # 1086 ; # 1094 ; # 1077 ; # 1089 ; # 1089 ; # 1086 ; # 1074 ; # 1087 ; # 1086 ; # 1075 ; # 1083 ; # 1086 ; # 1097 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1080 ; # 1080 ; # 1089 ; # 1087 ; # 1086 ; # 1083 ; # 1100 ; # 1079 ; # 1086 ; # 1074 ; # 1072 ; # 1085 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; . # 1052 ; # 1099 ; # 1087 ; # 1086 ; # 1083 ; # 1091 ; # 1095 ; # 1072 ; # 1077 ; # 1084 ; # 1080 ; # 1085 ; # 1092 ; # 1086 ; # 1088 ; # 1084 ; # 1072 ; # 1094 ; # 1080 ; # 1102 ; # 1080 ; # 1079 ; # 1090 ; # 1072 ; # 1082 ; # 1080 ; # 1093 ; # 1084 ; # 1077 ; # 1090 ; # 1086 ; # 1076 ; # 1072 ; # 1093 ; # 1082 ; # 1072 ; # 1082 ; # 1087 ; # 1086 ; # 1076 ; # 1075 ; # 1086 ; # 1090 ; # 1086 ; # 1074 ; # 1082 ; # 1072 ; # 1089 ; # 1099 ; # 1088 ; # 1099 ; # 1093 ; # 1087 ; # 1080 ; # 1097 ; # 1077 ; # 1074 ; # 1099 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1091 ; # 1082 ; # 1090 ; # 1086 ; # 1074 ; # 1076 ; # 1083 ; # 1103 ; # 1082 ; # 1091 ; # 1083 ; # 1080 ; # 1085 ; # 1072 ; # 1088 ; # 1080 ; # 1080 ; , # 1087 ; # 1086 ; # 1090 ; # 1088 ; # 1077 ; # 1073 ; # 1083 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; , # 1080 ; # 1083 ; # 1080 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; . # 1048 ; # 1079 ; # 1089 ; # 1 090 ; # 1072 ; # 1090 ; # 1077 ; # 1081 ; # 1084 ; # 1099 ; # 1090 ; # 1072 ; # 1082 ; # 1078 ; # 1077 ; # 1091 ; # 1079 ; # 1085 ; # 1072 ; # 1077 ; # 1084 ; # 1086 ; # 1073 ; # 1080 ; # 1089 ; # 1082 ; # 1091 ; # 1089 ; # 1089 ; # 1090 ; # 1074 ; # 1077 ; # 1086 ; # 1090 ; # 1073 ; # 1086 ; # 1088 ; # 1072 ; , # 1087 ; # 1086 ; # 1076 ; # 1075 ; # 1086 ; # 1090 ; # 1086 ; # 1074 ; # 1082 ; # 1080 ; , # 1080 ; # 1086 ; # 1073 ; # 1089 ; # 1083 ; # 1091 ; # 1078 ; # 1080 ; # 1074 ; # 1072 ; # 1085 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1077 ; # 1082 ; # 1088 ; # 1072 ; # 1089 ; # 1085 ; # 1099 ; # 1093 ; # 1087 ; # 1080 ; # 1097 ; # 1077 ; # 1074 ; # 1099 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1091 ; # 1082 ; # 1090 ; # 1086 ; # 1074 ; # 1080 ; # 1083 ; # 1080 ; # 1087 ; # 1080 ; # 1097 ; # 1077 ; # 1074 ; # 1099 ; # 1093 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1091 ; # 1082 ; # 1090 ; # 1086 ; # 1074 ; , # 1090 ; # 1088 ; # 1072 ; # 1076 ; # 1080 ; # 1094 ; # 1080 ; # 1086 ; # 1085 ; # 1085 ; # 1086 ; # 1086 ; # 1090 ; # 1085 ; # 1086 ; # 1089 ; # 1103 ; # 1097 ; # 1080 ; # 1093 ; # 1089 ; # 1103 ; # 1082 ; # 1088 ; # 1077 ; # 1075 ; # 1080 ; # 1086 ; # 1085 ; # 1091 ; # 1080 ; # 1083 ; # 1080 ; # 1085 ; # 1072 ; # 1089 ; # 1077 ; # 1083 ; # 1077 ; # 1085 ; # 1080 ; # 1102 ; . # 1058 ; # 1072 ; # 1082 ; # 1095 ; # 1090 ; # 1086 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1077 ; # 1080 ; # 1084 ; # 1077 ; # 1077 ; # 1090 ; # 1074 ; # 1072 ; # 1078 ; # 1085 ; # 1086 ; # 1077 ; # 1084 ; # 1077 ; # 1089 ; # 1090 ; # 1086 ; # 1074 ; # 1089 ; # 1092 ; # 1077 ; # 1088 ; # 1077 ; # 1077 ; # 1075 ; # 1086 ; # 1080 ; # 1079 ; # 1091 ; # 1095 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; . 3. # 1057 ; # 1090 ; # 1086 ; # 1095 ; # 1082 ; # 1080 ; # 1079 ; # 1088 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1091 ; # 1095 ; # 1077 ; # 1085 ; # 1099 ; # 1093 ; # 1089 ; # 1086 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; # 1089 ; # 1072 ; # 1084 ; # 1072 ; # 1103 ; # 1089 ; # 1077 ; # 1088 ; # 1100 ; # 1077 ; # 1079 ; # 1085 ; # 1072 ; # 1103 ; # 1087 ; # 1088 ; # 1086 ; # 1073 ; # 1083 ; # 1077 ; # 1084 ; # 1072 ; . # 1051 ; # 1102 ; # 1073 ; # 1086 ; # 1077 ; # 1084 ; # 1085 ; # 1086 ; # 1078 ; # 1077 ; # 1089 ; # 1090 ; # 1074 ; # 1086 ; # 1084 ; # 1077 ; # 1090 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1089 ; # 1086 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1079 ; # 1072 ; # 1097 ; # 1080 ; # 1097 ; # 1072 ; # 1077 ; # 1090 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1077 ; # 1086 ; # 1090 ; # 1087 ; # 1086 ; # 1088 ; # 1095 ; # 1080 ; # 1087 ; # 1086 ; # 1089 ; # 1083 ; # 1077 ; # 1091 ; # 1088 ; # 1086 ; # 1078 ; # 1072 ; # 1103 ; # 1080 ; # 1083 ; # 1080 ; # 1088 ; # 1077 ; # 1079 ; # 1085 ; # 1080 ; . 4. # 1055 ; # 1088 ; # 1072 ; # 1082 ; # 1090 ; # 1080 ; # 1082 ; # 1072 ; # 1089 ; # 1086 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; # 1080 ; # 1084 ; # 1077 ; # 1077 ; # 1090 ; # 1076 ; # 1086 ; # 1080 ; # 1089 ; # 1090 ; # 1086 ; # 1088 ; # 1080 ; # 1095 ; # 1077 ; # 1089 ; # 1082 ; # 1086 ; # 1077 ; # 1087 ; # 1088 ; # 1086 ; # 1080 ; # 1089 ; # 1093 ; # 1086 ; # 1078 ; # 1076 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; . # 1044 ; # 1088 ; # 1077 ; # 1074 ; # 1085 ; # 1080 ; # 1077 ; # 1083 ; # 1102 ; # 1076 ; # 1080 ; # 1074 ; # 1099 ; # 1089 ; # 1091 ; # 1096 ; # 1080 ; # 1074 ; # 1072 ; # 1083 ; # 1080 ; # 1087 ; # 1083 ; # 1086 ; # 1076 ; # 1099 ; # 1080 ; # 1086 ; # 1074 ; # 1086 ; # 1097 ; # 1080 ; , # 1074 ; # 1099 ; # 1078 ; # 1078 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; # 1077 ; # 1079 ; # 1077 ; # 1088 ; # 1085 ; # 1086 ; # 1093 ; # 1083 ; # 1077 ; # 1073 ; # 1085 ; # 1086 ; # 1075 ; # 1086 ; # 1079 ; # 1083 ; # 1072 ; # 1082 ; # 1072 ; , # 1089 ; # 1086 ; # 1083 ; # 1077 ; # 1085 ; # 1091 ; # 1102 ; # 1080 ; # 1074 ; # 1099 ; # 1089 ; # 1091 ; # 1096 ; # 1077 ; # 1085 ; # 1085 ; # 1091 ; # 1102 ; # 1088 ; # 1099 ; # 1073 ; # 1091 ; # 1080 ; # 1080 ; # 1075 ; # 1088 ; # 1091 ; . # 1069 ; # 1090 ; # 1080 ; # 1089 ; # 1090 ; # 1072 ; # 1088 ; # 1099 ; # 1077 ; # 1084 ; # 1077 ; # 1090 ; # 1086 ; # 1076 ; # 1099 ; # 1088 ; # 1072 ; # 1079 ; # 1074 ; # 1080 ; # 1074 ; # 1072 ; # 1083 ; # 1080 ; # 1089 ; # 1100 ; # 1086 ; # 1095 ; # 1077 ; # 1085 ; # 1100 ; # 1084 ; # 1077 ; # 1076 ; # 1083 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; , # 1090 ; # 1086 ; # 1075 ; # 1076 ; # 1072 ; # 1080 ; # 1084 ; # 1077 ; # 1083 ; # 1086 ; # 1084 ; # 1077 ; # 1089 ; # 1090 ; # 1086 ; # 1073 ; # 1099 ; # 1090 ; # 1100 ; # 1101 ; # 1084 ; # 1087 ; # 1080 ; # 1088 ; # 1080 ; # 1095 ; # 1077 ; # 1089 ; # 1082 ; # 1086 ; # 1077 ; # 1073 ; # 1088 ; # 1086 ; # 1078 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; , # 1074 ; # 1099 ; # 1089 ; # 1099 ; # 1093 ; # 1072 ; # 1085 ; # 1080 ; # 1077 ; , # 1082 ; # 1091 ; # 1088 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; , # 1080 ; # 1083 ; # 1077 ; # 1095 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; # 1089 ; # 1089 ; # 1086 ; # 1083 ; # 1100 ; # 1102 ; . 5. # 1047 ; # 1072 ; # 1090 ; # 1077 ; # 1084 ; # 1087 ; # 1086 ; # 1089 ; # 1083 ; # 1077 ; # 1076 ; # 1086 ; # 1074 ; # 1072 ; # 1083 ; # 1087 ; # 1088 ; # 1086 ; # 1075 ; # 1088 ; # 1077 ; # 1089 ; # 1089 ; . # 1058 ; # 1072 ; # 1082 ; # 1080 ; # 1084 ; # 1086 ; # 1073 ; # 1088 ; # 1072 ; # 1079 ; # 1086 ; # 1084 ; , # 1084 ; # 1099 ; # 1087 ; # 1088 ; # 1080 ; # 1086 ; # 1073 ; # 1088 ; # 1077 ; # 1090 ; # 1072 ; # 1077 ; # 1084 ; # 1080 ; # 1085 ; # 1092 ; # 1086 ; # 1088 ; # 1084 ; # 1072 ; # 1094 ; # 1080 ; # 1102 ; # 1086 ; # 1073 ; # 1072 ; # 1082 ; # 1090 ; # 1077 ; # 1088 ; # 1080 ; # 1103 ; # 1093 ; , # 1087 ; # 1086 ; # 1095 ; # 1074 ; # 1072 ; # 1093 ; , # 1080 ; # 1083 ; # 1080 ; # 1076 ; # 1088 ; # 1086 ; # 1078 ; # 1078 ; # 1072 ; # 1093 ; , # 1084 ; # 1080 ; # 1082 ; # 1088 ; # 1086 ; # 1086 ; # 1088 ; # 1075 ; # 1072 ; # 1085 ; # 1080 ; # 1079 ; # 1084 ; # 1072 ; # 10 93 ; . # 1054 ; # 1085 ; # 1080 ; # 1075 ; # 1083 ; # 1072 ; # 1074 ; # 1085 ; # 1099 ; # 1077 ; # 1087 ; # 1088 ; # 1080 ; # 1095 ; # 1080 ; # 1085 ; # 1099 ; # 1087 ; # 1086 ; # 1088 ; # 1095 ; # 1080 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; . # 1053 ; # 1086 ; # 1077 ; # 1089 ; # 1090 ; # 1100 ; # 1087 ; # 1088 ; # 1080 ; # 1073 ; # 1083 ; # 1080 ; # 1079 ; # 1080 ; # 1090 ; # 1077 ; # 1083 ; # 1100 ; # 1085 ; # 1086 ; # 1089 ; # 1077 ; # 1084 ; # 1100 ; # 1080 ; # 1079 ; # 1074 ; # 1077 ; # 1089 ; # 1090 ; # 1085 ; # 1099 ; # 1093 ; # 1084 ; # 1077 ; # 1090 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1074 ; # 1089 ; # 1086 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1080 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; # 1090 ; # 1077 ; # 1087 ; # 1077 ; # 1088 ; # 1100 ; , # 1090 ; # 1080 ; # 1087 ; # 1072 ; : # 1086 ; # 1093 ; # 1083 ; # 1072 ; # 1078 ; # 1076 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; # 1080 ; # 1083 ; # 1080 ; # 1079 ; # 1072 ; # 1084 ; # 1086 ; # 1088 ; # 1072 ; # 1078 ; # 1080 ; # 1074 ; # 1072 ; # 1085 ; # 1080 ; # 1077 ; , # 1086 ; # 1073 ; # 1077 ; # 1079 ; # 1074 ; # 1086 ; # 1078 ; # 1080 ; # 1074 ; # 1072 ; # 1085 ; # 1080 ; # 1077 ; , # 1082 ; # 1086 ; # 1085 ; # 1089 ; # 1077 ; # 1088 ; # 1074 ; # 1080 ; # 1088 ; # 1086 ; # 1074 ; # 1072 ; # 1085 ; # 1080 ; # 1077 ; , # 1082 ; # 1091 ; # 1088 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; , # 1089 ; # 1086 ; # 1083 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; , # 1080 ; # 1076 ; # 1086 ; # 1087 ; # 1086 ; # 1083 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; # 1093 ; # 1080 ; # 1084 ; # 1080 ; # 1095 ; # 1077 ; # 1089 ; # 1082 ; # 1080 ; # 1093 ; # 1082 ; # 1086 ; # 1085 ; # 1089 ; # 1077 ; # 1088 ; # 1074 ; # 1072 ; # 1085 ; # 1090 ; # 1086 ; # 1074 ; # 1080 ; # 1080 ; # 1085 ; # 1075 ; # 1080 ; # 1073 ; # 1080 ; # 1090 ; # 1086 ; # 1088 ; # 1086 ; # 1074 ; . # 1054 ; # 1095 ; # 1077 ; # 1085 ; # 1100 ; # 1095 ; # 1072 ; # 1089 ; # 1090 ; # 1086 ; # 1086 ; # 1073 ; # 1098 ; # 1077 ; # 1076 ; # 1080 ; # 1085 ; # 1103 ; # 1102 ; # 1090 ; # 1085 ; # 1077 ; # 1089 ; # 1082 ; # 1086 ; # 1083 ; # 1100 ; # 1082 ; # 1086 ; # 1087 ; # 1088 ; # 1080 ; # 1085 ; # 1094 ; # 1080 ; # 1087 ; # 1086 ; # 1074 ; . 6. # 1057 ; # 1086 ; # 1074 ; # 1088 ; # 1077 ; # 1084 ; # 1077 ; # 1085 ; # 1085 ; # 1072 ; # 1103 ; # 1094 ; # 1077 ; # 1083 ; # 1100 ; # 1089 ; # 1086 ; # 1093 ; # 1088 ; # 1072 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1103 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; # 1074 ; # 1082 ; # 1083 ; # 1102 ; # 1095 ; # 1072 ; # 1077 ; # 1090 ; # 1087 ; # 1088 ; # 1086 ; # 1080 ; # 1079 ; # 1074 ; # 1086 ; # 1076 ; # 1089 ; # 1090 ; # 1074 ; # 1086 ; # 1087 ; # 1086 ; # 1082 ; # 1072 ; # 1095 ; # 1077 ; # 1089 ; # 1090 ; # 1074 ; # 1091 ; # 1087 ; # 1088 ; # 1086 ; # 1076 ; # 1086 ; # 1074 ; # 1086 ; # 1083 ; # 1100 ; # 1089 ; # 1090 ; # 1074 ; # 1080 ; # 1103 ; , # 1087 ; # 1086 ; # 1101 ; # 1082 ; # 1086 ; # 1085 ; # 1086 ; # 1084 ; # 1080 ; # 1080 ; , # 1080 ; , # 1086 ; # 1089 ; # 1086 ; # 1073 ; # 1077 ; # 1085 ; # 1085 ; # 1086 ; , # 1076 ; # 1083 ; # 1103 ; # 1091 ; # 1076 ; # 1086 ; # 1073 ; # 1089 ; # 1090 ; # 1074 ; # 1072 ; # 1074 ; # 1076 ; # 1086 ; # 1087 ; # 1086 ; # 1083 ; # 1085 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; # 1082 ; # 1087 ; # 1088 ; # 1077 ; # 1076 ; # 1086 ; # 1090 ; # 1074 ; # 1088 ; # 1072 ; # 1097 ; # 1077 ; # 1085 ; # 1080 ; # 1102 ; # 1087 ; # 1086 ; # 1088 ; # 1095 ; # 1080 ; . # 1062 ; # 1074 ; # 1077 ; # 1090 ; # 1080 ; # 1083 ; # 1080 ; # 1087 ; # 1086 ; # 1103 ; # 1074 ; # 1083 ; # 1077 ; # 1085 ; # 1080 ; # 1077 ; , # 1072 ; # 1088 ; # 1086 ; # 1084 ; # 1072 ; # 1090 ; , # 1089 ; # 1090 ; # 1088 ; # 1091 ; # 1082 ; # 1090 ; # 1091 ; # 1088 ; # 1072 ; 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Monday, November 25, 2019

Free Essays on Internet Censorship

Internet Censorship The debate over whether the government should censor the Internet is intense. In 1995 the senate passed the Communications Decency Act written by Senator Jim Exon. The act â€Å"outlaws ‘obscene, lewd, lascivious, filthy or indecent’ communications on the Internet,† (Exon 130). Americans on both sides of the issue are asking some very pertinent, yet difficult to answer, questions. Does censorship of the Internet violate our First Amendment rights? Is regulation of the Internet even possible? Will censoring the Internet protect children from inappropriate material or will it hinder those searching for legitimate information? As a mother, I am concerned about my child having access to pornographic or otherwise inappropriate material on the Internet. However, my personal belief is that it is impossible to regulate the Internet without infringing on the liberties of the First Amendment. Simply put: one person’s definition of inappropriate or pornographic material may be totally different from another’s. It must be made clear that although I am neither totally for nor against censorship, I do not wish to have a limit set on what I can and cannot access determined by someone else’s values. Those who support censorship of Internet materials feel that applying obscenity laws to the internet will protect children from pornography without â€Å"significantly† infringing upon our First Amendment rights (Exon 125). Supporters of this view argue that we live with restrictions on our freedom of speech everyday, such as: libel laws and laws against false advertising. These people submit that the â€Å"anti-pornography laws that exist for U.S. mail, broadcast and telephone communications,† should also apply to computers (Exon 126). These people suggest the use of blocking software in libraries and support the required use of â€Å"a verified credit card, debit account, adult access code or personal identification number... Free Essays on Internet Censorship Free Essays on Internet Censorship Thesis: Government Censorship would damage the atmosphere of the freedom to express ideas on the Internet; therefore, government should not encourage censorship. Introduction I. In the Internet community, there is a large volume of technical terms. For this reason, it is first necessary to examine the terminology specific to Internet. 1.The internet is a world wide computer network. 1.Electronic mail (email), which is one component of the Internet, approximates person to person letters, memoranda, notes and even phone calls. 2.Another term that is often used is electronic news (enews/Usenet), enews is a broadcast, free to the Internet medium. 3.The term FTP is also frequently used. File transfer protocol (FTP) started as an Internet archival and retrieval medium, somewhat analogous to traditional libraries. 4.The world-wide web (WWW), which is another component of the Net, can be used to "publish" material that would traditionally appear in journals, magazines, posters, books, television and even on film. 2.It is also essential to give a brief history on the internet. 3.The U.S. government is now trying to pass bills to prevent misuse of the Net. II. In order to understand the need for the ever-growing body of legislation, it is important to explore the controversy, and the current problems involved with the Net as it exists must be introduced. 1.The problem that concerns most people is offensive materials such as pornography. 2.Another crucial internet crime is the stealing of credit card numbers. III. One reaction to this inapplicability has been the "Censor the Net" approach (the censorship bill), we are now to compare its advantages and disadvantages. 1.First, the meaning of "Censoring the Net" must be explained. 2.However, many experts have pointed out that government censorship is not possible. 1.First, it is not fair to exclude the freedom and damage the atmosphere of... Free Essays on Internet Censorship Freedom of Speech: Censorship of the Internet Many of use it daily. We find it useful, and it has become part of our everyday lives. â€Å"It† is the Internet. The Internet has dramatically changed our society. It brings together people and their ideas from all around the world in a short amount of time. It is expanding daily to allow new ideas and thought s to be transmitted quickly and easily with the single click of a button. One can find information on almost any subject there. Yet many people are trying to censor it. The Internet is accessed by millions of people around the world each day. If the Internet is to considered a global resource it must remain uncensored. Is Internet Censorship Needed? Internet censorship seems to be the target of many debates nowadays in the U.S. due to the rising popularity of the Internet and the large amounts of pornography, warez, illegal drugs, and general threats to society. It is a very hard subject to handle, after all no individual is in charge of the internet, and in fact no one really owns it except perhaps the â€Å"millions of people throughout the world who contribute to it in various ways† (Burton). The argument for censorship has been going on for at least 5 years now and no one sees an answer being had anytime soon. Getting rid of all the offensive content on the Internet would perhaps make it more productive but is it legal to stifle the rights of others like that? The government thought so at one point and tried to pass a law to help filter the Internet only to have it found unconstitutional soon after To understand why the Internet is subject to such a controversial debate, we must first learn what it is and what it contains. The Internet is a method of communication and a source of information that is becoming more popular among those who are interested in, and have the time to surf the information superhighway. The Internet has been in universal use for many years. It... Free Essays on Internet Censorship Internet Censorship The debate over whether the government should censor the Internet is intense. In 1995 the senate passed the Communications Decency Act written by Senator Jim Exon. The act â€Å"outlaws ‘obscene, lewd, lascivious, filthy or indecent’ communications on the Internet,† (Exon 130). Americans on both sides of the issue are asking some very pertinent, yet difficult to answer, questions. Does censorship of the Internet violate our First Amendment rights? Is regulation of the Internet even possible? Will censoring the Internet protect children from inappropriate material or will it hinder those searching for legitimate information? As a mother, I am concerned about my child having access to pornographic or otherwise inappropriate material on the Internet. However, my personal belief is that it is impossible to regulate the Internet without infringing on the liberties of the First Amendment. Simply put: one person’s definition of inappropriate or pornographic material may be totally different from another’s. It must be made clear that although I am neither totally for nor against censorship, I do not wish to have a limit set on what I can and cannot access determined by someone else’s values. Those who support censorship of Internet materials feel that applying obscenity laws to the internet will protect children from pornography without â€Å"significantly† infringing upon our First Amendment rights (Exon 125). Supporters of this view argue that we live with restrictions on our freedom of speech everyday, such as: libel laws and laws against false advertising. These people submit that the â€Å"anti-pornography laws that exist for U.S. mail, broadcast and telephone communications,† should also apply to computers (Exon 126). These people suggest the use of blocking software in libraries and support the required use of â€Å"a verified credit card, debit account, adult access code or personal identification number... Free Essays on Internet Censorship The Internet is a wonderful place of entertainment and education but like all places used by millions of people, it has some murky corners people would prefer children not to explore. In the physical world society as a whole conspires to protect children, but there are no social or physical constraints to Internet surfing. The Internet Censorship Bill of 1995, also known as the Exon/Coats Communications Decency Act, has been introduced in the U.S. Congress. It would make it a criminal offense to make available to children anything that is indecent, or to send anything indecent with "intent to annoy, abuse, threaten, or harass" ("Stop the Communications ..." n.p.). The goal of this bill as written (though not as stated by its proponents) is to try to make all public discourse on the Internet suitable for young children. The issue of whether is it necessary to have censorship on the Internet is being argued all over the world. There are numerous homepages on the World Wide Web discussing this issue, or asking people to sign the petition to stop government censorship. The Internet was originally a place for people to freely express their ideas worldwide. It is also one of America's most valuable types of technology; scientists use email for quick and easy communication. They post their current scientific discoveries on the Usenet newsgroups so other scientists in the same field of study all over the world can know in minutes. Ordinary people use the Net for communication, expressing their opinions in the newsgroups, obtaining up-to-date information from the WWW, acquiring files by using FTP, etc. Censorship would damage the atmosphere of the freedom to express ideas on the Internet; therefore, government should not encourage censorship. In the Internet community, there is a large volume of technical terms. For this reason, it is first necessary to examine the terminology specific to Internet. The Internet is a world wide computer net...

Friday, November 22, 2019

Google - Financial Statement Analysis Research Paper

Google - Financial Statement Analysis - Research Paper Example In September 2002, Google News was released with over 4000 news sources (google.com, 2010). Google Scholar was released a month later with Google maps quickly following in February 2005. In late 2005, the Google Reader and Analytics products were released. Adding to the list of company innovations include such popular items as Google Video, Google Docs, Google Gmail, Google Investor, Google Images, and Google Picassa (google.com, 2010). The purchase of YouTube was a major purchase in 2006 where it gave the public access to making their own videos and sharing them with other online users (google.com, 2010). Besides YouTube, the purchase of Doubleclick was a major purchase where Google attempted to further enhance and simplify how advertisements are bought and sold over the internet (edgaronline, 2010). The Android product is a new portable phone that the company recognizes as needed to compete in a mobile world that we live in. The company wishes to have mobile devices and applications as an important building block for the future (google.com, 2010). Marketing efforts have been relatively low cost for the company by use of public relations and simple customers sharing experiences to spread the word that Google offers a quick and efficient service along with useful products (edgaronline, 2010). The company’s sales and support department attempts to highlight the advantages of products like AdSense to leading companies all over the globe. Although customers sign up online for products such as AdSense, the sales team attempts to build positive working relationships and promote key product advantages to other companies (edgaronline, 2010). The current environment for Google is a highly competitive one which is always changing. The company faces potential threats from: (1) other search companies; (2) social networking companies; (3) new and emerging mobile products; and (4) specialized search and e-commerce sites (edgaronline,

Wednesday, November 20, 2019

Fundamentals of Macroeconomics paper Research Example | Topics and Well Written Essays - 500 words

Fundamentals of Macroeconomics - Research Paper Example Real domestic product however defines the value of a country’s total production in goods and services at constant base prices of the commodities. Such a basis may be each commodity’s price in a given year (Mankiw, 2011). Purchasing of groceries reduces amount of money among the purchasing households and increases amount of money held by businesses. It also increases government revenues through tax on the groceries. It however has no economic value on households and business because it involves exchange of similar values. The government however derives economic gain in the taxes (Mankiw, 2011). The layoff reduces households’ revenues because of its unemployment effect on members of households. It also adversely affects businesses through reduced revenues because of households’ lower spending power. Layoffs also affect the government through lost revenues in income tax. The government’s revenue is further reduced because lower purchasing power into reduced demand results in low revenues from value added tax (Mankiw, 2011). Decrease in tax has the effect of increasing households’ money reserves from both direct and indirect taxes. A decrease in direct tax results in higher income and a decrease in indirect taxes leads to lower commodity prices that allows for more savings by households. The effects of reduced tax on households also mean their increased purchasing power that suggests an increase in sales and revenues among businesses. The government however losses revenues that it would however earn at higher taxes (Mankiw, 2011). Purchasing groceries has no net economic flow between households and businesses because it is a mere exchange of equivalent values between the two parties. Money, however, flows from both businesses and households to the government in taxes (Mankiw, 2011). Lowering taxes identifies a virtual transfer of

Monday, November 18, 2019

Polyvinylchloride Contraversy Essay Example | Topics and Well Written Essays - 500 words

Polyvinylchloride Contraversy - Essay Example The main characteristics of PVC include:Excellent thermal and sound insulation: PVC has extremely low thermal conductivity provides superior heat insulation. This makes it suitable for use in windows and electric cables among others.PVC is flexible in design: It can be utilized safely in numerous circumstances.PVC requires no maintenance: It is not affected by atmospheric agents because it is manufactured from stable materials.PVC is flame retardant: It is self-extinguishing thus used in construction and all other areas of application.The characteristics of PVC make it suitable for many uses. It is used in:Building and construction: PVC is widely used in building and construction. It is used for windows, doors, pipes, shutters, frames and tiles among others (Joyce, 2005).Automobile: PVC is used to manufacture various car components such as dashboards, doors and electrical cabinets among others.Electricity and electronics: PVC is used to make electric cables for both domestic and indu strial use, pipes and sockets among other electric appliances. Packaging: PVC is commonly used to package foodstuffs as well as pharmaceuticals (Rapra Technology Limited, 2 007).Manufacturing or vinyl toys.Despite its extensive use and low cost, PVC has not been without controversy over its safety. Research has shown that high amounts of phthalates are toxic to human health. Earl Gray revealed in his work that male reproductive development is highly responsive to certain phthalates.

Saturday, November 16, 2019

Serum Urate Concentrations and the Risk of Hyperuricemia

Serum Urate Concentrations and the Risk of Hyperuricemia Common UCP2 variants contribute to serum urate concentrations and the risk of hyperuricemia Luyu Yang, Zheng Dong, Jingru zhou, Yanyun Ma, Weilin Pu, Dongbao Zhao, Hongjun He, Hengdong Ji, Yajun Yang, Xiaofeng Wang, Xia Xu, Yafei Pang, Hejian Zou,  Li Jin,  Chengde Yang*, Jiucun Wang* *Corresponding author These authors equally contributed to this study.   Abstract Elevated serum urate, which is regulated at multiple levels including genetic variants, is a risk factor for gout and other metabolic diseases. This study aimed to investigate the association between UCP2 variants and serum urate as well as hyperuricemia in a Chinese population. In total, 4332 individuals were genotyped for two common UCP2 variants, -866G/A and Ala55Val. These loci were not associated either serum urate level or with a risk of hyperuricemia in the total group of subjects. However, in females, -866G/A and Ala55Val were associated with a lower serum urate (P = 0.006 and 0.014à ¯Ã‚ ¼Ã…’seperately) and played a protective role against hyperuricemia (OR = 0.80, P = 0.018; OR = 0.79, P = 0.016). These associations were not observed in the males. After further stratification, the two loci were associated with serum urate in overweight, but not underweight females. The haplotype A-T (-866G/A-Ala55Val) was a protective factor for hyperuricemia in the female subgroup (OR = 0.80, P=0.017). This present study identified a novel gene, UCP2, that influences the serum urate concentration and the risk of hyperuricemia, and the degree of association varies with gender and BMI levels.   Introduction Uric acid is the final product of purine oxidation in humans. Elevated serum urate, or hyperuricemia, has long been recognized as an independent risk factor for gout [1-2]. There is a renewed interest in hyperuricemia and its association with a number of other clinical disorders including hypertension, atherosclerosis, cardiovascular disease, chronic kidney diseases, and abdominal obesity, glucose intolerance, insulin resistance, and dyslipidemia, which are often subsumed under the term metabolic syndrome [3]. Serum urate is balanced between uric acid production in the liver and its disposal via the kidney and gut [4]. The occurrence of hyperuricemia could be caused by disruptions in any part of this metabolic process. Both genetic and environmental factors, such as gender and body mass index (BMI), have a strong effect on the risk of hyperuricemia [3]. Among those factors, the attribution of genetic factors is estimated to be as high as 73% [5]. Recent genome-wide association studies (GWAS) have identified 28 loci associated with serum urate concentration [6]. However, only approximately 7% of the variation in serum urate concentration could be explained by those reported loci, suggesting the missing heritability remained to be explored [6]. Human uncoupling proteins (UCPs) are mitochondrial transporters present in the inner membrane of mitochondria [7]. UCPs are capable of uncoupling ATP production from mitochondrial respiration by causing proton leak and preventing mitochondrial hyperpolarization and the formation of reactive oxygen species (ROS) [8]. Among the five identified UCPs, UCP2 is widely expressed in almost all mammalian tissues including white adipose tissue, liver, kidney, pancreatic islets, macrophages and retinal endothelial cells, indicating its involvement in a variety of physiologic or pathologic events [9-12]. Two of the most common polymorphisms of this gene, -866G/A (rs659366) in the promoter and Ala55Val (rs660339) in codon 55, were identified as being associated with different phenotypes [7, 12], including obesity, insulin resistance, type 2 diabetes mellitus (T2D), low-density lipoprotein (LDL) particle size, coronary incidence and other metabolic disorders [9-10, 13-21]. Given the involvement of UCP2 and hyperuricemia in a variety of metabolic disorders, we selected the two common loci -866G/A and Ala55Val to explore the association between genetic UCP2 variants and hyperuricemia in a Chinese population, offering a new diagnostic or therapeutic target for hyperuricemia. Results There was no  association between SNPs and serum urate The two loci were proven in Hardy-Weinberg equilibrium (-866G/A: P = 0.990; Ala55Val: P = 0.690). For -866G/A, AA, AG, and GG genotypes accounted for 21.6%, 49.9%, and 28.6% of hyperuricemic patients, respectively; in healthy controls, the distribution was 21.2%, 49.6%, and 29.3%, respectively. As shown in Table 1, the -866G/A polymorphism was not found to be associated with serum urate (AA/GG: Beta = -0.008, P = 0.644; AG/GG:Beta = -0.012, P = 0.474) or with the risk of hyperuricemia (AA/GG: OR = 1.05, P = 0.603; AG/GG:OR = 1.03, P = 0.667). For Ala55Val, the TT, TC, and CC genotype distribution was 21.5%, 50.5% and 28.0% in hyperuricemic patients, respectively, and the distribution was 21.5%, 49.8% and 28.6% in healthy controls, respectively. No association was observed between Ala55Val polymorphism and serum urate (TT/CC: Beta = -0.013, P = 0.460; TC/CC:Beta = -0.017, P = 0.324). There was no difference in the distribution of the genotypes or alleles among hyperuricemic patients a nd healthy controls (TT/CC: OR = 1.02, P = 0.824; TC/CC:OR = 1.04, P = 0.652). Therefore, no statistically solid evidence supported the genetic effect of -866G/A and Ala55Val on serum urate or the risk of hyperuricemia in the total group of subjects. UCP2 variants were associated withserum urate andhyperuricemia in female subgroups As shown in Table 1, we stratified all subjects into male and female subgroups to further explore the gender-related genetic effects of the two polymorphisms. In the male subgroups, there were no significant associations between the two loci and serum urate or the risk of hyperuricemia (all P > 0.025). However, some nominal significant associations were found between -866G/A and the hyperuricemia risk (genotype AA: OR = 1.26, P = 0.038; allele A: OR = 1.12, P = 0.035), indicating a possible risky effect of the -866G/A variant on hyperuricemia incidence in males. A significant association was found between SNPs and serum urate and hyperuricemia in the female subgroups. The -866G/A genotypes were associated with a lower serum urate (AA/GG: Beta = -0.078, P = 0.015; AG/GG: Beta = -0.104, P = 0.001) and a decreased risk of hyperuricemia (AG/GG: OR = 0.71, P = 0.025). The subjects carrying allele A had a lower serum urate and a decreased risk of hyperuricemia (A/G: Beta = -0.054, P = 0.006; OR = 0.80, P = 0.018). For Ala55Val, genotype TT carriers showed a lower serum urate (TT/CC: Beta = -0.075, P = 0.022) and a decreased risk of hyperuricemia (TT/CC: OR = 0.64, P = 0.020). Genotype TC carriers only had a lower serum urate (TC/CC: Beta = -0.082, P = 0.012) but no decreased risk of hyperuricemia (TC/CC: OR = 0.77, P = 0.093). Allele T was associated with a lower serum urate (T/C: Beta = -0.049, P = 0.016) and a decreased risk of hyperuricemia (T/C: OR = 0.79, P = 0.016). Further analysis of associationin females  with different BMI levels Further analysis was performed regarding the genetic effect of UCP2 variants on serum urate and the risk of hyperuricemia among females with different BMI levels (Table 2). The majority of the females enrolled were stratified into normal- or overweight group (Table 2). In the underweight subgroup, whose sample size was limited after stratification, no significant association was observed between the two loci and serum urate or hyperuricemia risk (all P > 0.025, Table 2). In the normal weight subgroup, -866G/A genotype AA+AG carriers were associated with a lower serum urate (AA+AG/GG: Beta = -0.095, P = 0.022) but not with a decreased risk of hyperuricemia (AA+AG/GG: OR = 0.65, P = 0.076). However, the Ala55Val genotypes or alleles showed no statistical association with serum urate (TT+TC/CC: Beta = -0.070, P = 0.091; T/C: Beta = -0.047, P = 0.106) or hyperuricemia (TT+TC/CC: OR = 0.72, P = 0.173; T/C: OR = 0.72, P = 0.051). In the overweight subgroup, the genotypes of both loci were associated a lower serum urate (AA+AG/GG: Beta = -0.138, P = 0.001; TT+TC/CC: Beta = -0.130, P = 0.003) and a significant, or at least marginal, decreased risk of hyperuricemia (AA+AG/GG: OR = 0.62, P = 0.015; TT+TC/CC: OR = 0.74, P = 0.027). However, the alleles of the loci were associated with a lower serum urate level (A/G: Beta = -0.072, P = 0.019; T/C: Beta = -0.072, P = 0.019) but not with a decreased risk of hyperuricemia (A/G: OR = 0.75, P = 0.036; T/C: OR = 0.74, P = 0.027). Our results suggested a stronger effect of UCP2 variants on overweight females than on normal weight females (Table 2). Association between haplotypes and risk of hyperuricemia As listed in Table 3, the haplotypes of the two loci were estimated in the total group of subjects and after stratification by gender. The -866G/A and Ala55Val variants were in strong linkage disequilibrium (D = 0.974, r2 = 0.936). The wild type haplotype G-C (-866G/A-Ala55Val) was applied as the reference one. Haplotype A-T made up for the most frequent one, while single mutation at -866G/A or Ala55Val each accounted for less than 1 percent (Table 3). In the total group of subjects, no haplotypes were correlated with susceptibility of hyperuricemia. In the female subgroups, haplotype A-T (-866G/A-Ala55Val) was associated with a decreased risk of hyperuricemia; however, this association was null in males. No further significant associations between hyperuricemia and other two rare haplotypes were found in our study, partly due to the limited size of the rare haplotypes carriers (Table 3). These results correlated with the association between genotypes or alleles and hyperuricemia (Ta ble 1). Discussion Uncoupling protein 2 (UCP2) is present in the inner mitochondrial membrane and mainly decreases the ATP level and ROS produced by electron transport; therefore, UCP2 is involved in a board range of pathological processes. In the present study, we first focused on the relationship between UCP2 variants and serum urate and hyperuricemia, potentially examining the scope of the loci related to hyperuricemia. The present study revealed no association between the two polymorphisms of UCP2 and serum urate or hyperuricemia in the total group of subjects. However, because serum urate is extensively influenced by gender differences, we stratified the total group of subjects and determined that -866G/A and Ala55Val were associated with serum urate and hyperuricemia in females [25-26]. Females with the -866G/A genotype AA+AG or allele A had lower serum urate and a decreased risk of hyperuricemia, indicating a protective role of -866G/A for hyperuricemia in females. The -866G/A variant is a functional polymorphism located in the promoter region and putatively changes the transcription factor binding sites [7]. The wild type G allele in -866G/A was associated with lower UCP2 mRNA expression [19, 27]. Increased UCP2 mRNA expression from the A allele was translated into an increased amount of UCP2 protein, with corresponding induced proton leak, decreased ATP/ADP ratio and enhanced elimination of ROS [10, 19]. Hypermethylation in the promoter region could affect the binding of transcripation factors, causing aberrant gene expression. Consistent with our expectations, we found a typical CpG island in the UCP2 promoter region, which included the locus of the -866G/A variant, using information from the University of California-Santa Cruz (UCSC; Santa Cruz, CA, USA) database (http://genome.ucsc.edu/cgi-bin/hgGateway). We believe the UCP2 promoter variant -866G/A could shape this CpG island and protect the UCP2 promoter region from DNA methylation, unco vering a novel underlying mechanism that determines -866G/A increases UCP2 transcription. Uric acid accumulation is caused by the acceleration of ATP degradation to AMP, a precursor of uric acid, and UCP2 could decrease the ATP level and lower redundant AMP for uric acid formation [7, 28]. Moreover, an elevation of serum urate concentration occurs as a physiologic response to increased oxidative stress [31]. Because the ROS level could be down-regulated by UCP2, a counter-regulatory increase of serum urate as an antioxidant defense is less urgent. Therefore, the -866G/A variant in the promoter region might serve as a protective factor through a higher UCP2 mRNA level and increased translation of the UCP2 protein, which might regulate ROS and modify the ATP/ADP ratio. The other locus, Ala55Val, is a missense variant in exon 4 and is associated with an altered degree of uncoupling [7]. In our study, a protective effect for hyperuricemia and lower serum urate were observed in genotype TT and allele T in the female subgroups. However, the genetic effect of the Ala55Val variant was less clear. Several researchers identified an association of Ala55Val with the BMI level and type 2 diabetes mellitus (T2D), with controversial conclusions within cohorts, and few functional studies were performed [14, 32-33]. Similar to -866G/A, the protective role of the Ala55Val variant for hyperuricemia might also be attributed to altered UCP2 transcription. In the male subgroups, a less statistically significant but possible effect of -866G/A and Ala55Val was observed for hyperuricemia risk and higher serum urate. Similar gender-associated genetic effects of UCP2 variants were more or less observed for diseases other than hyperuricemia [7]. For example, Heidema et al. suggested a genetic effect of UCP2 on weight gain was regulated through different mechanism in males and females [34]. Lee, et al. demonstrated that the association between UCP2 variants and BMI was more apparent among female subjects [35]. Cheurfa et al. confirmed the association of UCP2 variants with coronary artery diseases in males but not females [36]. In the present study, we found UCP2 variants -866G/A and Ala55Val had a stronger effect on females with hyperuricemia. One possible explanation for the gender-associated genetic effects of UCP2 might be a regulation role of sex hormones such as estrogen. Estrogen was reported to repress UCP2 in a breast cancer cell line and papillary thyroid cancer cells [37-38]. Taken together, these results suggest the UCP2 protein level was down-regulated by estrogen in females but reversed by the variants of -866G/A and Ala55Val, providing a plausible explanation for the specific protective effects of UCP2 variants on females [37]. Genetic effects on hyperuricemia and obesity have been widely recognized [3]. In the present study, we found that -866G/A and Ala55Val were associated with lower serum urate and a decreased risk of hyperuricemia in overweight, but not underweight, females (Table 2). The relative small sample size might limit the correlation analysis in the underweight group. However, we did observe females with higher BMI level were more likely to benefit from the protective genetic effect of -866G/A and Ala55Val, where the association was significant between the two SNPs and serum urate level of risk of hyperuricemia. In the contrast, among the normal weight females, -866G/A, but not Ala55Val, showed a significant association with a low risk of hyperuricemia, indicating a less contribution from the protective effect of UCP2 variants than seen in overweight females. It was also implied from our results that the [tw1]functional à ¢Ã‹â€ Ã¢â‚¬â„¢866G>A promoter variant displayed a stronger effect. The interactions between obesity, uric acid and UCP2 were complicated. BMI has long been viewed as an essential factor influencing uric acid [3]. UCP2 transcription was activated by fatty acids [16]. A recent meta-analysis revealed that UCP2 -866G/A and Ala55Val are associated with a risk of obesity [32]. Subtle intermediary obesity related phenotypes such as elevated triglycerides, total cholesterol concentrations, increased the risk of dyslipidemia and circulating leptin levels were also observed to be correlated with UCP2 variants [40]. Based on these results, we assumed lipid metabolism material such as fatty acids participated in and enhanced the genetic effect of UCP2 variants on serum urate regulation, explaining the stronger genetic effect of UCP2 variants on females with higher BMI levels observed in the present study. The -866G/A and Ala55Val variants were in strong linkage disequilibrium (D = 0.974, r2 = 0.936). The haplotype frequency analysis revealed that variants of the two loci were more in co-variant haplotype A-T (-866G/A-Ala55Val) compared with the single variant forms of G-T or A-C (Table 3). Haplotype A-T was associated with a decreased risk of hyperuricemia only in females, which was consistent with the genotype or alleles results. However, the small size of the two rare haplotypes might limit the power of association analysis with hyperuricemia risk to a certain extent. The susceptibility of hyperuricemia in the two rare haplotype carriers required validation in a larger cohort. Conclusion The present study identified a novel gene, UCP2, with two loci, -866G/A and Ala55Val; this gene influenced the serum urate concentrations and the risk of hyperuricemia in females. The associations of those loci were affected by gender and BMI. This study supported the potential involvement of this gene in the prevention, prediction and treatment of hyperuricemia. Materials and methods Experimental design A total of 4332 subjects were enrolled from the Taizhou Longitudinal Study [22] and included 1387 hyperuricemic patients and 2945 healthy controls. The associations of common UCP2 variants with serum urate and hyperuricemia were tested by linear regression and logistic regression with or without gender stratification, respectively. A body mass index (BMI) subgroup was also used for further analysis. Participants All subjects were enrolled from Taizhou Longitudinal Study [22], of which 1387 individuals had serum urate level over 7 mg/dl and were treated as hyperuricemic patients, and 2945 individuals had normal serum urate (à ¢Ã¢â‚¬ °Ã‚ ¤ 7 mg/dl) and were treated as healthy controls [23]. The subjects were divided into subgroups (underweight: BMI à ¯Ã¢â€š ¬Ã‚ ¼ 18.5; normal weight: 18.50 à ¯Ã¢â‚¬Å¡Ã‚ £ BMI Genetic analysis Genetic analysis was carried out in accordance with the written informed consent and guideline offered by the Ethical Committees of the School of Life Science of Fudan University. For genetic analysis, peripheral blood was collected from all the individuals included in this study. Genomic DNA was extracted from whole blood using the QIAamp DNA Blood Mini kit (QIAGEN, Germany) and was stored at -20à ¢Ã¢â‚¬Å¾Ã†â€™. The DNA concentration and quality (including optical density (OD) 260/280 and 260/230 measurements) were determined using a Nanodrop Lite spectrophotometer (Thermo Fisher Scientific, Waltham, MA, USA). Genotyping of -866G/A and Ala55Val in UCP2 were performed by SNPscan according to the manufacturers instructions. Statistical analysis The clinical characteristics were presented as the mean à ¯Ã¢â‚¬Å¡Ã‚ ± SD. Students t-test was used to test for a significant difference in the mean age, BMI and serum urate between hyperuricemic patients and healthy controls. The chi-square test was used to describe the gender distribution difference between hyperuricemic patients and healthy controls. The chi-square test was used to test Hardy-Weinberg equilibrium (HWE) of the two loci. We conducted a logistic regression analysis to calculate adjusted odd ratio (OR) with 95% confidence interval (95% CI) and P-values to describe the distribution of -866G/A and Ala55Val adjusted for age and gender between hyperuricemic patients and healthy controls. A linear regression was performed to calculate Beta and P-values to estimate the effect on serum urate in different genotypes and alleles. Genotype GG, allele G of -866G/A and genotype CC, allele C of Ala55Val were used as references, respectively. Stratification into subgroups was performed on the basis of gender and different BMI values for further analysis. Haplotype frequencies between the hyperuricemic patients and controls were estimated by OR (95% CI) and chi-square test. The haplotype of the most frequent (-866G/A-Ala55Val, G-C) was used as the reference. A 2-sided P-value less than 0.025 was considered statistically significant after multiple correlation by Bonferroni method. The PHASE program (V2.1) was used for haplotype frequencies estimation, and SPSS 19.0 was used for the statistical analysis. References 1. Choi HK, Mount DB, Reginato AM. Pathogenesis of gout. Ann Intern Med 2005;143(7):499-516. 2.  Weaver AL. Epidemiology of gout. Cleve Clin J Med 2008;75 Suppl 5:S9-12. 3.  Billiet L, Doaty S, Katz JD, Velasquez MT. Review of hyperuricemia as new marker for metabolic syndrome. ISRN Rheumatol 2014;2014:852954. 4.  Hediger MA, Johnson RJ, Miyazaki H, Endou H. Molecular physiology of urate transport. Physiology (Bethesda) 2005;20:125-33. 5.  Kolz M, Johnson T, Sanna S, Teumer A, Vitart V, Perola M, et al.. Meta-analysis of 28,141 individuals identifies common variants within five new loci that influence uric acid concentrations. PLoS Genet 2009;5(6):e1000504. 6.  Kà ¶ttgen A, Albrecht E, Teumer A, Vitart V, Krumsiek J, Hundertmark C, et al.. Genome-wide association analyses identify 18 new loci associated with serum urate concentrations. Nature Genetics 2012;45(2):145-154. 7.  Donadelli M, Dando I, Fiorini C, Palmieri M. UCP2, a mitochondrial protein regulated at multiple levels. In; 2014. p. 1171-90. 8.  Azzu V, Jastroch M, Divakaruni AS, Brand MD. The regulation and turnover of mitochondrial uncoupling proteins. Biochimica et Biophysica Acta (BBA) Bioenergetics 2010;1797(6-7):785-791. 9.  Dalgaard LT, Andersen G, Larsen LH, Sorensen TI, Andersen T, Drivsholm T, et al.. Mutational analysis of the UCP2 core promoter and relationships of variants with   obesity. Obes Res 2003;11(11):1420-7. 10. Dalgaard LT. Genetic Variance in Uncoupling Protein 2 in Relation to Obesity, Type 2 Diabetes, and Related Metabolic Traits: Focus on the Functional -866G>A Promoter Variant (rs659366). J Obes 2011;2011:340241. 11. Rousset S, Mozo J, Dujardin G, Emre Y, Masscheleyn S, Ricquier D, et al.. UCP2 is a mitochondrial transporter with an unusual very short half-life. FEBS Lett 2007;581(3):479-82. 12. Jarmuszkiewicz W, Woyda-Ploszczyca A. [Mitochondrial uncoupling proteins: regulation and physiological role]. Postepy Biochem 2008;54(2):179-87. 13. Yu X, Wieczorek S, Franke A, Yin H, Pierer M, Sina C, et al.. Association of UCP2 -866 G/A polymorphism with chronic inflammatory diseases. In; 2009. p. 601-5. 14. Rosmond R, Bouchard C, Bjorntorp P. Lack of association between the uncoupling protein-2 Ala55Val gene polymorphism and phenotypic features of the Metabolic Syndrome. Biochim Biophys Acta 2002;1588(2):103-5. 15.  Heidari J, Akrami SM, Heshmat R, Amiri P, Fakhrzadeh H, Pajouhi M. Association study of the -866G/A UCP2 gene promoter polymorphism with type 2 diabetes and obesity in a Tehran population: a case control study. Arch Iran Med 2010;13(5):384-90. 16.  Emre Y, Nà ¼bel T. Uncoupling protein UCP2: When mitochondrial activity meets immunity. In; 2010. p. 1437-1442. 17. Toda C, Diano S. Mitochondrial UCP2 in the central regulation of metabolism. Best Pract Res Clin Endocrinol Metab 2014;28(5):757-64. 18. Shen Y, Wen Z, Wang N, Zheng Z, Liu K, Xia X, et al.. Investigation of variants in UCP2 in Chinese type 2 diabetes and diabetic retinopathy. PLoS One 2014;9(11):e112670. 19. Sesti G, Cardellini M, Marini MA, Frontoni S, DAdamo M, Del GS, et al.. A common polymorphism in the promoter of UCP2 contributes to the variation in insulin secretion in glucose-tolerant subjects. Diabetes 2003;52(5):1280-3. 20.  Otaegui D, Saenz A, Ruiz-Martinez J, Olaskoaga J, Lopez DMA. UCP2 and mitochondrial haplogroups as a multiple sclerosis risk factor. In; 2007. p. 454-8. 21.  Oktavianthi S, Trimarsanto H, Febinia CA, Suastika K, Saraswati MR, Dwipayana Pà ¦Ã¢â‚¬ ºÃ‚ ´AW, et al.. Uncoupling protein 2 gene polymorphisms are associated with obesity. Cardiovascular diabetology 2012;11(1):41. 22.  Wang X, Lu M, Qian J, Yang Y, Li S, Lu D, et al.. Rationales, design and recruitment of the Taizhou Longitudinal Study. BMC Public Health 2009;9:223. 23.  Mandell BF. Clinical manifestations of hyperuricemia and gout. Cleve Clin J Med 2008;75 Suppl 5:S5-8. 24.  WHO Global Database on Body Mass Index (BMI): an interactive surveillance tool for monitoring nutrition transition. Public Health Nutr. 2006; 9(5):658-. doi:10.1079/Phn2006967. ISI:000239972300019.. 25. Terkeltaub RA. Clinical practice. Gout. N Engl J Med 2003;349(17):1647-55. 26.  Neogi T. Clinical practice. Gout. N Engl J Med 2011;364(5):443-52.   Ã‚   27.  Lapice E, Pinelli M, Pisu E, Monticelli A, Gambino R, Pagano G, et al.. Uncoupling protein 2 G(-866)A polymorphism: a new gene polymorphism associated with C-reactive protein in type 2 diabetic patients. Cardiovasc Diabetol 2010;9:68. 28.  Richette P, Bardin T. Gout. Lancet 2010;375(9711):318-28. 29. Waring WS, Webb DJ, Maxwell SR. Systemic uric acid administration increases serum antioxidant capacity in healthy volunteers. J Cardiovasc Pharmacol 2001;38(3):365-71. 30. Zhang M, Wang M, Zhao Z. Uncoupling protein 2 gene polymorphisms in association with overweight and obesity susceptibility: A meta-analysis. Meta Gene 2014;2:143-159. 31.Vogler S, Goedde R, Miterski B, Gold R, Kroner A, Koczan D, et al.. Association of a common polymorphism in the promoter of UCP2 with susceptibility to multiple sclerosis. J Mol Med (Berl) 2005;83(10):806-11. 32.Heidema AG, Wang P, van Rossum CT, Feskens EJ, Boer JM, Bouwman FG, et al.. Sex-specific effects of CNTF, IL6 and UCP2 polymorphisms on weight gain. Physiol Behav 2010;99(1):1-7. 33.Lee YH, Kim W, Yu BC, Park BL, Kim LH, Shin HD. Association of the ins/del polymorphisms of uncoupling protein 2 (UCP2) with BMI   in a Korean population. Biochem Biophys Res Commun 2008;371(4):767-71. 34.Cheurfa N, Dubois-Laforgue D, Ferrarezi DA, Reis AF, Brenner GM, Bouche C, et al.. The common -866G>A variant in the promoter of UCP2 is associated with decreased risk of coronary artery disease in type 2 diabetic men. Diabetes 2008;57(4):1063-8. 35.Nadal-Serrano M, Sastre-Serra J, Pons DG, Miro AM, Oliver J, Roca P. The ERalpha/ERbeta ratio determines oxidative stress in breast cancer cell lines in response to 17beta-estradiol. J Cell Biochem 2012;113(10):3178-85. 36.Hima S, Sreeja S. Regulatory role of estrogen-induced reactive oxygen species in the modulatory function of UCP 2 in papillary thyroid cancer cells. IUBMB Life 2015. 37.Hamada T, Kotani K, Fujiwara S, Sano Y, Domichi M, Tsuzaki K, et al.. The UCP2-866 A/A genotype is associated with low density lipoprotein particle sizes in the general population. Med Sci Monit 2008;14(3):CR107-11. Acknowledgements This research was supported by grants from the Science and Technology Committee of Shanghai Municipality (11DJ1400100), International ST Cooperation Program of China (2013DFA30870), Ministry of Science and Technology (2011BAI09B00), and Program for 2012 Outstanding Medical Academic Leader for Hejian Zou. The computations involved in this study were supported by Fudan University High-End Computing Center. Author contributions statement

Wednesday, November 13, 2019

Prison: Inside and Out Essay -- Criminal Justice

Freedom in any society is a condition that is directly correlated to the compliance of society’s rules, regulations, and laws. This right afforded to citizens under the constitution is surrendered by those who choose to disregard and trample on the set of standards and behavior needed for a society to maintain order and avoid anarchy. When loss of freedom is the tool used to punish this segment of society, prisons become the instrumentality used to carry out the various freedom restrictions under the law for each individual. Citizens who find themselves incarcerated in prison will be exposed to an entirely different environment than those in free society. In addition, there is a psychological impact on those being reintroduced into society after having completed long-term sentences, an impact many believe contribute to the high recidivism rates in the United States. The criminal justice system with all of its connected inner workings may not address all of the concerns inside and out of our current prison system, but just as we have improved on our prisons from the past, we will continue to improve and implement new techniques and design precise programs in our future prisons. The saying, crime does not pay, probably held more significance a century ago when the judicial system and prison officials focused on the idea that severe punishment was the best deterrent to crime. Punishment in prisons was more than just loss of freedom, it involved chain gangs, hard-labor, the hole, and other inhumane treatment designed to ward off the would be criminals and beat the current criminals into submission. Today, Jeremy Bentham’s theory that punishment must outweigh the benefits of crime is addressed only through loss of freedoms. Prison... ...I Law Enforcement Bulletin: http://www.fbi.gov/stats-services/publications/law-enforcement-bulletin/july-2010/cell-phones-as-prison-contraband Coley, R. J., & Barton, P. E. (2006). Lock Up and Locked Out: An Educational Perspective on the U.S. Prison Population. Princeton New Jersey: Policy Evaluation and Research Center. Retrieved April 28, 2012, from http://www.ets.org/Media/Research/pdf/PIC-LOCKEDUP.pdf Hanson, D. J. (2010). Effectiveness of D.A.R.E. Retrieved from Alcohol Problems and Solutions: http://www2.potsdam.edu/hansondj/youthissues/1059145293.html McDonnell, B.. (2011). Improving Public Safety through Prisoner Reentry Programs. Ripon Forum, 45(2), 6-7. Retrieved April 29, 2012, from ProQuest Social Science Journals. (Document ID: 2387305821). Seiter, R. P. (2011). Corrections an Introduction. Upper Saddle River, N.J.: Pearson Education.

Monday, November 11, 2019

Old Story Time

The story Emma based from the book â€Å"A World of Prose† and published by the author Carolyn Cole is about a woman named Emma who is a very beautiful lady of leisure, she has stepped down from an active social life after giving birth to Dorian in order to spend more time with her daughter Maria. Her husband Jack is a notorious cheater and he is not faithful to Emma. The setting of the story occurs in three places the York Residence, an unknown mall and at the old train station where the death of Emma took place.In the first setting of the story which takes place at the York Residence shows the two friends Maria and Dorian doing activities such as chatting about Dory’s Mother Emma, playing adult games and the support of each other. The moods and the themes displayed in that setting of the story is called Innocence which is epitomized by Dorian, the story is told from her perspective therefore we get a view of the innocence behind her misunderstanding of adult conversat ions and situations.Friendship which is shown in the friendship between Dorian and Maria, which is characterized by playing adult games. In the second setting which takes place at the unnamed mall shows Dory, Maria Mrs Robinson and Emma going to the mall to buy school cloths for Dorian because the summer was ending, while there the two friends Dory and Maria played games in the arcade while Mrs Robinson and Emma was having close conversations about sending both Maria and Dorian to a boarding school and about Emma having another child but Emma denied her because her husband Jack was not ready for Dory.While they were chatting, Dory and Maria wanted to go ice skating at the old train station and Emma decides to send them, the girls got their skates Emma hugged Maria before they left. The moods and themes displayed in that setting is called Love and family Relationship, there are two types of families in the short story, the nuclear and the single family. Dorian’s family is the nuclear family, consisting of mother, father and child. Maria’s family is the single family with her single mother Mrs Robinson.In the third or last setting which takes place at the old station shows Dory and Maria skating with the other kids at the old train station when the two girls caught a glimpse of the lady sitting on the steps wearing a black dress and long whit beads Maria pulled Dorian behind an old boxcar and Dory and Maria realises that it was her when they see their father with her they quickly assume that she was the lady at the train station. Maria belittles Dory quit a lot, and Dory indeed believes Maria to know and understand everything.But the game was over when Emma and Mrs Robinson arrive on the train and Emma experiences the mysterious lady with her husband she sadly sobs as she runs feeling cheated by own husband, she ran waiting to cross Georgia Avenue her husband grabbed her by her arm. She snatched away from him and ran into the street where an oncomi ng vehicle overruns her. The moods and themes displayed in that setting is called Innocence which is from Dory the story is told from her perspective therefore we get a view of the innocence behind her misunderstanding of adult conversations.

Saturday, November 9, 2019

Its all about life Essay

Life is beautiful but not always easy, it has problems, too, and the challenge lies in facing them with courage, letting the beauty of life act like a balm, which makes the pain bearable, during trying times, by providing hope Happiness, sorrow, victory, defeat, day-night are the two sides of the me coin. Similarly life is full of moments of joy, pleasure, success and comfort punctuated by misery, defeat, failures and problems. There is no human being on Earth, strong, powerful, wise or rich, who has not experienced, struggle, suffering or failure. No doubt, life is beautiful and every moment – a celebration of being alive, but one should be always ready to face adversity and challenges. A person who has not encountered difficulties in life can never achieve success. Difficulties test the courage, patience, perseverance and true character of a human being. Adversity and hardships make a person strong and ready to face the challenges of life with equanimity. There is no doubt that there can be no gain without pain. It is only when one toils and sweats it out that success is nourished and sustained. Thus, life is and should not be just a bed of roses; thorns are also a part of it and should be accepted by us just as we accept the beautiful side of life. The thorns remind one of how success and happiness can be evasive and thus not to feel disappointed and disheartened rather remember that the pain of thorns is short-lived, and the beauty of life would soon overcome the prick of thorns. Those, who are under the impression that life is a bed of roses are disillusioned soon and become victims of depression and frustration. One who faces difficulties with courage and accepts success without letting it go to its head is the one who experience real happiness, contentment and peace in life. Those, who think, that good times last forever, easily succumb to pressure during difficulties. They do not put in required hard work and efforts because they break down easily. You can take the example of a student, who burns the mid night oil, makes sacrifices and resists temptations so that he can perform well. Similarly, a successful executive has to face the ups and downs of life, not forgetting that life is a mix of success and failure, joy and sorrow. If he loses hope during difficult times, he would not achieve success and would be replaced by others. Even the strongest Kings and Emperors have had their cup of woes. Life has not been a bed of roses for them. The adage ‘Uneasy lays the head that wears the crown’ has been rightly used for people, who are successful and are enjoying power and authority. To sum up, life is beautiful just as roses but it has challenges which are like thorns and have to be faced and overcome by all. Those, who accept these, challenges and succeed, are the ones, who know how to live life in its true sense. Thus, enjoy life but also be prepared to bear the pricks of pain. Read more:Â  Bed of Roses Meaning

Wednesday, November 6, 2019

In Search of Peace essays

In Search of Peace essays What the world needs today is a civilization based on love and compassion, and not on competition. A civilization that is not dying to be victorious, but which is keen to live with peace. Nowadays terrorism, war and human killing are in vogue. Powerful nations control the less powerful nations as their right. Humanity is suffering at the hands of brutal politicians. There is no peace in the world and within the self. Innocent children are asking their parents, teachers and friends what is going on around them. Everybody is in search of peace. Where is peace? The world religions long ago defined peace as individual responsibility for the wellbeing of others. Let us remind ourselves of that forgotten lesson and reflect on how everyone of us individually can contribute towards peace in the world? Let us briefly survey the concept of peace given by the world religions. Peace means freedom from war and unrest. In ancient Hebrew thought, peace was not only the absence of war, but also the wellbeing, if not prosperity, of people. A famous passage which appears twice in the Bible (Is: 2-4 Mi. 4: 1-3) describes "... all nations are going to Jerusalem to learn the divine law; beating their swords into plowshares and their spear into pruning hooks, abandoning their swords and learning war no more." The study of Israelite traditions shows that peace was a social concept. It was visible and produced harmonious relationship in the family, in local society and between nations. The salutation shalom expressed positive aim of encouraging friendship, cooperation and living together for mutual benefit. The New Testament defines peace as "an orderly obedience to the eternal laws of God", so peace in the God's city is the perfect union of hearts in the enjoyment of God (19:13). The salutation of peace frequently appears in the New Testament. In the Quran, the Arabic word Salam has been used for peace. It has been generally used as a greet...

Monday, November 4, 2019

Biomedical Ethics Essay Example | Topics and Well Written Essays - 1750 words

Biomedical Ethics - Essay Example This paper is going to discuss and analyse the dignity of human life in relation the different concepts, views and ideas proposed by some philosophers and scientists. Looking at the video ‘The Island’, significant science fiction themes are clearly echoed in todays culture of medical ethics, class conflict and organ transplantation. Corporate ethics and prison populations are also reflected in the video. The main theme in the video is corporate abuse of human beings. Starred by Ewan McGregor, the video slowly builds up the theme and displays how the wealthy class engages in immoral activities in the hope that, they might be turned into immortals. By not fearing to destroy their vital body organs by engaging in drug abuse, excessive drinking, smoking and eating junk foodstuffs, these people show how wealth can be used to manipulate the good values and morality in humanity. The Island is an organization in the contemporary corporate world that makes its clients believe that it produces entirely new body organs in the laboratory yet this is not the case. It instead clones the clients and grows exact human beings whose body parts will be used in case of transplantations. The video is a true reflection of what is taking place in the contemporary world. The wealthy people and organizations are abusing corporate ethics in order to fulfil their desires. According to Kateb (2011), some doctors, including the highly revered Harry Sharp believed that social failure was a medical problem. This was in support of the scientific Eugenics movement sought to do away with some vices and immoral acts among human beings. Poverty, alcoholism, prostitution and criminal behaviour were the core vices that they sought to eliminate. Others included schizophrenia, rebelliousness and weak mindedness. Heated debates and arguments have however emerged concerning some of these

Saturday, November 2, 2019

A Detailed Analysis of the OpenOffice Research Paper

A Detailed Analysis of the OpenOffice - Research Paper Example In addition, the twin licensing system outlines that we are permitted to select either license. Moreover, both the LGPL and SISSL are OSI accepted as open source system licenses (OpenOffice1, 2012; OpenOffice, 2010). Application Similar to all other open source software applications, OpenOffice can be used as a commercial tool. In fact, as compared to other freeware applications, open source software is not restricted to only educational or personal use. In addition, there is no limit on where or how we utilize this software. Additionally, there are numerous applications of OpenOffice software. In this scenario, one of the key applications is Word processor application for creating documents. It also includes Spreadsheet-based application that is similar to MS Excel. Moreover, it also contains a Presentation Package for the creation of presentations. Furthermore, similar to MS Access, OpenOffice also offers database management system capability. As well, one of the most popular tools that OpenOffice contains is a drawing tool that is missing in traditional MS Office Package (OpenOffice1, 2012; OpenOffice, 2010). Goals OpenOffice.org is going to launch a 3rd version of the OpenOffice in the market, which is the result of over 20 years' software engineering. Intended from the initiate as a single element of software, it has reliability other products cannot compete. In addition, a totally open development procedure outlines that anybody is able to report bugs, can add new and innovative features, or improve the software. Moreover, OpenOffice.org version 3 offers everything that we need from our office software to carry out, the way we want to use it (OpenOffice1, 2012; OpenOffice, 2010).  Similar to all other open source software applications, OpenOffice can be used as a commercial tool. In fact, as compared to other freeware applications, open source software is not restricted to only educational or personal use. In addition, there is no limit on where or how we utilize this software. Additionally, there are numerous applications of OpenOffice software.Â